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Vitamin E para Atherosclerosis

C Evidências Evidência limitada de estudos de pequena dimensão, mas existem alguns sinais positivos.

Strong mechanistic rationale (LDL oxidation inhibition) but large RCTs (HOPE, GISSI-Prevenzione) show no benefit. Meta-analysis suggests potential harm at >=400 IU/day. AHA does not recommend supplementation.

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C

Conclusão

Strong mechanistic rationale (LDL oxidation inhibition) but large RCTs (HOPE, GISSI-Prevenzione) show no benefit. Meta-analysis suggests potential harm at >=400 IU/day. AHA does not recommend supplementation.

Key Study Findings

Review
Navigating the Effects of Anti-Atherosclerotic Supplements and Acknowledging Associated Bleeding Risks.
Dose: None vs: None Outcome: None Efeito: None None

População: Review of anti-atherosclerotic supplements and bleeding

Other
Extensive Bioactivity of Astaxanthin from Haematococcus pluvialis in Human.
Dose: None vs: None Outcome: None Efeito: None None

População: None

Other
Supplementation with Resveratrol, Piperine and Alpha-Tocopherol Decreases Chronic Inflammation in a Cluster of Older Adults …
Dose: None vs: Conventional treatment Outcome: effect of a dietary supplement on chronic inflammation … Efeito: None p < 0.05

População: None

Review
Nutrient intake, nutritional status, and cognitive function with aging.
Dose: None vs: None Outcome: None Efeito: None None

População: Aging adults (nutrition and cognition)

Review
Role of reactive oxygen species and antioxidants in atopic dermatitis.
Dose: None vs: None Outcome: Role of oxidative stress in atopic dermatitis Efeito: None None

População: Patients with atopic dermatitis

Review
Astaxanthin: a novel potential treatment for oxidative stress and inflammation in cardiovascular disease.
Dose: None vs: None Outcome: Astaxanthin: a novel potential treatment for oxidative stress … Efeito: None None

População: None

Key Statistics

15

Estudos

50000

Participantes

Mixed

C

Nota

Referenced Papers

Dosage & Usage

mg = milligrams · mcg = micrograms (1,000× smaller) · IU = International Units

Dosagens Comumente Utilizadas

note:
High-dose supplementation no longer recommended based on clinical trial evidence
general:
15 mg/day (22.4 IU)

Limite superior: 1,000 mg/day (1,500 IU natural, 1,100 IU synthetic)

Dosagens Estudadas em Pesquisas

Dosagem Duração Efeito N
None -- Mixed --
None -- Positive --
None -- Mixed --
None -- Mixed --
None -- Positive --
None -- Positive --

Melhor horário: With meals containing fat

Safety & Side Effects

Efeitos Colaterais Relatados

  • Increased bleeding risk at high doses
  • Possible increased all-cause mortality at >=400 IU/day
  • Increased heart failure hospitalization risk (HOPE/HOPE-TOO)
  • Nausea, diarrhea, fatigue at high doses

Interações Conhecidas

  • Anticoagulants (increased bleeding risk)
  • Statins and niacin (may blunt HDL-raising effect)
  • Chemotherapy and radiation (theoretical interference with oxidative mechanisms)
  • Vitamin K (may antagonize at high doses)

Ingestão máxima tolerável: 1,000 mg/day (1,500 IU natural, 1,100 IU synthetic)

Consulte sempre o seu profissional de saúde antes de iniciar qualquer suplemento.Sempre consulte seu profissional de saúde antes de iniciar qualquer suplemento.

Frequently Asked Questions

Does Vitamin E help with Atherosclerosis?
Based on 15 studies with 50,000 participants, there is limited but promising evidence that Vitamin E may support Atherosclerosis management. Our evidence grade is C (Some Evidence).
How much Vitamin E should I take for Atherosclerosis?
Studies have used various dosages. A commonly studied range is High-dose supplementation no longer recommended based on clinical trial evidence. Always consult your healthcare provider before starting any supplement regimen.
Are there side effects of Vitamin E?
Reported side effects may include Increased bleeding risk at high doses, Possible increased all-cause mortality at >=400 IU/day, Increased heart failure hospitalization risk (HOPE/HOPE-TOO), Nausea, diarrhea, fatigue at high doses. Most side effects are mild and dose-dependent. Consult your doctor if you experience any adverse reactions.
How strong is the evidence for Vitamin E and Atherosclerosis?
We rate the evidence as Grade C (Some Evidence). This rating is based on 15 peer-reviewed studies with 50,000 total participants. The overall direction of effect is mixed.

References

  1. [1] Maria-Zinaida Dobre et al.. Int J Mol Sci. 2025. Navigating the Effects of Anti-Atherosclerotic Supplements and Acknowledging Associated Bleeding Risks. doi:10.3390/ijms262010183 PubMed
  2. [2] Eiji Yamashita. Adv Exp Med Biol. 2021. Extensive Bioactivity of Astaxanthin from Haematococcus pluvialis in Human. doi:10.1007/978-981-15-7360-6_23 PubMed
  3. [3] Raúl Francisco Pastor et al.. Nutrients. 2020. Supplementation with Resveratrol, Piperine and Alpha-Tocopherol Decreases Chronic Inflammation in a Cluster of Older Adults with Metabolic Syndrome. doi:10.3390/nu12103149 PubMed
  4. [4] Katherine L Tucker. Ann N Y Acad Sci. 2016. Nutrient intake, nutritional status, and cognitive function with aging. doi:10.1111/nyas.13062 PubMed
  5. [5] N Sivaranjani et al.. J Clin Diagn Res. 2013. Role of reactive oxygen species and antioxidants in atopic dermatitis. doi:10.7860/JCDR/2013/6635.3732 PubMed
  6. [6] Fredric J Pashkow et al.. Am J Cardiol. 2008. Astaxanthin: a novel potential treatment for oxidative stress and inflammation in cardiovascular disease. doi:10.1016/j.amjcard.2008.02.010 PubMed

Aviso Legal da FDA: Estas declarações não foram avaliadas pela Food and Drug Administration. Os produtos e informações neste site não se destinam a diagnosticar, tratar, curar ou prevenir qualquer doença. As notas de evidência apresentadas são baseadas em nossa análise de pesquisas revisadas por pares publicadas e não constituem aconselhamento médico. Sempre consulte seu profissional de saúde antes de iniciar qualquer regime de suplementação.