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76 査読済み研究からの図表

すべて Coenzyme Q10 (CoQ10) Vitamin D Folate (Vitamin B9) Magnesium Potassium Calcium Selenium Resveratrol Quercetin Lycopene Berberine
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Figure 1. Blood pressure measurement (tail-cuff). Each bar represents values expressed as mean ± SEM. The change in mean arterial pressure (MAP) was considered significant (*) when p < 0.05 between two groups. The data were analyzed using two-way ANOVA
Figure 6 Chart

Blood pressure measurements by tail-cuff method showing significant changes in mean arterial pressure across treatment groups. Chronic metabolic acidosis elicited a hypertensive response, supporting the hypothesis that sustained acid-base imbalance directly contributes to elevated blood pressure.

Chronic Metabolic Acidosis Elicits Hypertension via Upregulation of Intrarenal Angiotensin II and …

Figure 7
Figure 7 Chart

Renal tissue analysis or additional hemodynamic data from the chronic metabolic acidosis hypertension study. The intrarenal measurements help delineate the local versus systemic mechanisms driving the blood pressure response.

Chronic Metabolic Acidosis Elicits Hypertension via Upregulation of Intrarenal Angiotensin II and …

Figure 8
Figure 8 Chart

Molecular or biochemical analysis of kidney tissue from animals with chronic metabolic acidosis. The data examine expression of inducible nitric oxide synthase and angiotensin pathway components in the renal cortex.

Chronic Metabolic Acidosis Elicits Hypertension via Upregulation of Intrarenal Angiotensin II and …

Figure 5. Urinary sodium analysis. Each bar represents values expressed as mean ± SEM. The change in urine sodium levels were considered significant (*) when p < 0.05 between groups. The data were analyzed using one-way ANOVA followed by Tukey’s multip
Figure 9 Chart

Urinary sodium excretion analysis across experimental groups reveals altered renal sodium handling during chronic metabolic acidosis. Changes in sodium reabsorption may contribute to the volume-dependent component of acidosis-induced hypertension.

Chronic Metabolic Acidosis Elicits Hypertension via Upregulation of Intrarenal Angiotensin II and …

Figure 6. Serum sodium and potassium levels. Each bar represents values expressed as mean ± SEM. (A) The changes in serum sodium levels were considered significant when * p < 0.05 (control vs. CMA) groups and # p < 0.05 (CMA vs. CMA + captopril or CM
Figure 10 Chart

Serum sodium and potassium levels measured during chronic metabolic acidosis. Electrolyte imbalances, particularly in potassium homeostasis, are closely linked to both acid-base disturbances and blood pressure regulation through renal and vascular mechanisms.

Chronic Metabolic Acidosis Elicits Hypertension via Upregulation of Intrarenal Angiotensin II and …

Figure 8. Plasma aldosterone (A) and plasma renin (B) analysis. Each bar represents values expressed as mean ± SEM. The changes in plasma aldosterone and renin were considered significant when * p < 0.05 (control vs. CMA) groups. The data were analyzed
Figure 11 Chart

Plasma aldosterone and plasma renin measurements indicate activation of the systemic renin-angiotensin-aldosterone system during chronic metabolic acidosis. Elevated aldosterone levels suggest enhanced sodium retention and potassium excretion as contributors to the hypertensive phenotype.

Chronic Metabolic Acidosis Elicits Hypertension via Upregulation of Intrarenal Angiotensin II and …

Figure 12
Figure 12

Chronic Metabolic Acidosis Elicits Hypertension via Upregulation of Intrarenal Angiotensin II and …

Figure 13
Figure 13 Chart

Measured parameters from a study evaluating chronic Metabolic Acidosis Elicits Hypertension via Upregulation of Intrarenal Angiotensin II and Induction of Oxidative Stress, contributing to the overall assessment of chronic metabolic acidosis (CMA) can be a consequence of persistent hypertension but could potentially play a role in.

Chronic Metabolic Acidosis Elicits Hypertension via Upregulation of Intrarenal Angiotensin II and …

Figure 13. Schematic representation of possible pathophysiological routes of acidosis-induced increased blood pressure.
Figure 14 Diagram

Schematic representation of possible pathophysiological routes of acidosis-induced increased blood pressure.

Chronic Metabolic Acidosis Elicits Hypertension via Upregulation of Intrarenal Angiotensin II and …

Figure 2
Figure 2 Chart

Statistical analysis from research investigating dietary supplements and prevention of preeclampsia, comparing treatment groups and control conditions.

Dietary supplements and prevention of preeclampsia.

Fig. 1 Dietary reference intakes. Schematic diagram illustrating the risks of nutrient inadequacy and adverse effects
Figure 3 Diagram

Fig. 1 Dietary reference intakes.

Dietary supplements and prevention of preeclampsia.

Figure 1. Chemical structure and mechanisms of action of quercetin. Quercetin exerts many mechanisms of action, including anti-allergic, anti-viral, antioxidant, and anti-inflammatory activities. For more details, see the text and Table 1.
Figure 1 Diagram

Chemical structure of quercetin and illustration of its mechanisms of action, including anti-inflammatory, antioxidant, and immunomodulatory pathways relevant to allergic conditions.

Quercetin and Its Lecithin-Based Formulation: Potential Applications for Allergic Diseases Based on …

Figure 2
Figure 2 Chart

Pharmacological or clinical data supporting quercetin's potential applications for allergic rhinitis, asthma, or related inflammatory conditions.

Quercetin and Its Lecithin-Based Formulation: Potential Applications for Allergic Diseases Based on …

Figure 3
Figure 3 Chart

Comparative efficacy data for quercetin in different formulations, examining the impact of delivery systems on therapeutic outcomes.

Quercetin and Its Lecithin-Based Formulation: Potential Applications for Allergic Diseases Based on …

Figure 3. Plasma concentration of quercetin and its lecithin-based formulation. This pharmacokinetics study compared two groups of healthy volunteers supplemented with quercetin or quercetin phospholipids. Details are reported in the text. Partly modified
Figure 4 Chart

Plasma concentration curves comparing standard quercetin and its lecithin-based (phytosomal) formulation, demonstrating enhanced oral bioavailability of the lipid-based delivery system.

Quercetin and Its Lecithin-Based Formulation: Potential Applications for Allergic Diseases Based on …

Figure 1
Figure 1

Comparison of Coenzyme Q10 (Ubiquinone) and Reduced Coenzyme Q10 (Ubiquinol) as Supplement …

Figure 2
Figure 2

Comparison of Coenzyme Q10 (Ubiquinone) and Reduced Coenzyme Q10 (Ubiquinol) as Supplement …

Molecular docking Common enriched
Figure 1

Molecular docking Common enriched

Network Pharmacology and Molecular Docking Study of Yupingfeng Powder in the Treatment …

Figure 2
Figure 2

Network Pharmacology and Molecular Docking Study of Yupingfeng Powder in the Treatment …

Figure 3
Figure 3

Network Pharmacology and Molecular Docking Study of Yupingfeng Powder in the Treatment …

(d)
Figure 4

(d)

Network Pharmacology and Molecular Docking Study of Yupingfeng Powder in the Treatment …

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Figure 5

Network Pharmacology and Molecular Docking Study of Yupingfeng Powder in the Treatment …

Figure 6
Figure 6

Network Pharmacology and Molecular Docking Study of Yupingfeng Powder in the Treatment …

Figure 8: Sankey diagram of hub genes and enriched signaling pathways.
Figure 11

Figure 8: Sankey diagram of hub genes and enriched signaling pathways.

Network Pharmacology and Molecular Docking Study of Yupingfeng Powder in the Treatment …

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