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Figure 3

Descripción

Moderate hyperhomocysteinemia resulting from low folate status is an independent risk factor for cardiovascular disease, dementia, and depression. This figure maps the clinical consequences of impaired folate metabolism due to genetic variants.

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Population-level variation in folate-related genetic polymorphisms influences disease susceptibility across ethnic groups. This figure compares allele frequencies and associated health outcomes for key folate metabolism gene variants.

Figure 4

Population-level variation in folate-related genetic polymorphisms influences disease susceptibility across ethnic groups. This figure compares allele frequencies and associated health outcomes for key folate metabolism gene variants.

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Figure 3

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Source Paper

Genetic polymorphisms and folate status.

Congenital anomalies (2017)

PMID: 28598562

DOI: 10.1111/cga.12232

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![Figure 3: Moderate hyperhomocysteinemia resulting from low folate status is an independent risk factor for cardiovascular disease, dementia, and depression. This figure maps the clinical consequences of impaired folate metabolism due to genetic variants.](https://pdfs.citedhealth.com/figures/28598562/102.png)

> Source: Mami Hiraoka et al. "Genetic polymorphisms and folate status.." *Congenital anomalies*, 2017. PMID: [28598562](https://pubmed.ncbi.nlm.nih.gov/28598562/)
<figure>
  <img src="https://pdfs.citedhealth.com/figures/28598562/102.png" alt="Moderate hyperhomocysteinemia resulting from low folate status is an independent risk factor for cardiovascular disease, dementia, and depression. This figure maps the clinical consequences of impaired folate metabolism due to genetic variants." />
  <figcaption>Figure 3. Moderate hyperhomocysteinemia resulting from low folate status is an independent risk factor for cardiovascular disease, dementia, and depression. This figure maps the clinical consequences of impaired folate metabolism due to genetic variants.<br>  Source: Mami Hiraoka et al. "Genetic polymorphisms and folate status.." <em>Congenital anomalies</em>, 2017. PMID: <a href="https://pubmed.ncbi.nlm.nih.gov/28598562/">28598562</a></figcaption>
</figure>

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